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USMLEQA formerly USMLEFastTrack
United States
Приєднався 18 січ 2013
USMLEQA.com - Pass the USMLE Step 1 with Confidence: Master the Exam with Question & Answer based Learning
Відео
Ketone Bodies: An In-Depth Review in a Question and Answer Format
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usmleqa.com/?p=27654 Question: What are ketone bodies produced from in the liver? Answer: In the liver, fatty acids and amino acids are metabolized to produce acetoacetate and β-hydroxybutyrate.Question: What is the significance of oxaloacetate in relation to ketone bodies? Answer: In prolonged starvation and diabetic ketoacidosis, oxaloacetate is depleted for gluconeogenesis. With chronic alco...
Mastering the Pyruvate Dehydrogenase Complex: A Comprehensive Review
Переглядів 195Рік тому
usmleqa.com/?p=9444 Question: What is the Pyruvate dehydrogenase complex? Answer: The Pyruvate dehydrogenase complex is a mitochondrial enzyme complex that links glycolysis and the TCA cycle. It is differentially regulated in fed (active) and fasting (inactive) states.Question: What is the reaction catalyzed by the Pyruvate dehydrogenase complex? Answer: The Pyruvate dehydrogenase complex catal...
Glycolysis: A Comprehensive Q&A Review
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usmleqa.com/?p=9440 Question: What is the regulation of glycolysis? Answer: The regulation of glycolysis is controlled by a variety of enzymes and molecules, including ATP, glucose, and glucose-6-phosphate.Question: What are the key enzymes in glycolysis? Answer: The key enzymes in glycolysis include hexokinase or glucokinase, phosphofructokinase-1, phosphoglycerate kinase, and pyruvate kinase....
Familial Dyslipidemias - Type IV Hypertriglyceridemia: A Comprehensive Overview (Q&A Format)
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usmleqa.com/?p=27802 Question: What is Familial Dyslipidemias - Type IV? Answer: It is a type of hypertriglyceridemia.Question: How is Familial Dyslipidemias - Type IV inherited? Answer: It is inherited as an autosomal dominant trait.Question: What is the pathogenesis of Familial Dyslipidemias - Type IV? Answer: It is caused by hepatic overproduction of very low-density lipoprotein (VLDL).Quest...
In-Depth Review of Familial Dyslipidemias Type III: Dysbetalipoproteinemia - A Q&A Format
Переглядів 139Рік тому
usmleqa.com/?p=27800 Question: What is Familial Dyslipidemias? Answer: Familial Dyslipidemias is a type of genetic disorder that affects lipid metabolism.Question: What type of Familial Dyslipidemias is being discussed in the text? Answer: The text is discussing Familial Dyslipidemias Type III, also known as Dysbetalipoproteinemia.Question: How is Dysbetalipoproteinemia inherited? Answer: Dysbe...
Comprehensive Review of Familial Dyslipidemias - Type II Hypercholesterolemia: A Q&A Approach
Переглядів 109Рік тому
usmleqa.com/?p=27798 Question: What type of dyslipidemia is discussed in the text? Answer: Familial dyslipidemias - Type II HypercholesterolemiaQuestion: What is the mode of inheritance for Type II Hypercholesterolemia? Answer: The mode of inheritance is autosomal dominant (AD).Question: What is the pathogenesis of Type II Hypercholesterolemia? Answer: The pathogenesis is either due to absent o...
In-Depth Review of Familial Dyslipidemias -Type I: Questions and Answers
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usmleqa.com/?p=27744 Question: What is Familial Dyslipidemias -Type I? Answer: Familial Dyslipidemias -Type I is also known as Hyperchylomicronemia.Question: How is Familial Dyslipidemias -Type I inherited? Answer: Familial Dyslipidemias -Type I is inherited as an Autosomal Recessive trait.Question: What is the pathogenesis of Familial Dyslipidemias -Type I? Answer: The pathogenesis of Familial...
Comprehensive Review of Abetalipoproteinemia: Q&A Format for USMLE Step 1 Preparation
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usmleqa.com/?p=27743 "Question: What is Abetalipoproteinemia? Answer: Abetalipoproteinemia is a genetic disorder caused by a mutation in the gene that encodes microsomal transfer protein (MTP).Question: What is the mode of inheritance for Abetalipoproteinemia? Answer: Abetalipoproteinemia is an autosomal recessive disorder.Question: What lipoproteins are absent in Abetalipoproteinemia? Answer: ...
Lipoprotein Functions: A Comprehensive Review in Question and Answer Format
Переглядів 99Рік тому
usmleqa.com/?p=27741 Question: What are Lipoproteins composed of? Answer: Lipoproteins are composed of varying proportions of proteins, cholesterol, TGs (triglycerides), and phospholipids.Question: Which lipoprotein carries the most cholesterol? Answer: LDL (low-density lipoprotein) and HDL (high-density lipoprotein) carry the most cholesterol.Question: What is the importance of cholesterol? An...
Mastering Major Apolipoproteins: A Q&A Review for USMLE Step 1
Переглядів 73Рік тому
usmleqa.com/?p=27738 Question: What are the major apolipoproteins? Answer: The major apolipoproteins include Chylomicron, VLDL, IDL, LDL, and HDL.Question: What is the function of Chylomicron? Answer: Chylomicron mediates the uptake of remnants, everything except LDL.Question: What is the function of AI found only on alpha-lipoproteins (HDL)? Answer: AI found only on alpha-lipoproteins (HDL) ac...
A Comprehensive Review of Key Enzymes in Lipid Transport for USMLE Step 1
Переглядів 116Рік тому
usmleqa.com/?p=27736 Question: What is Cholesteryl Ester Transfer Protein (CETP)? Answer: CETP mediates the transfer of cholesteryl esters to other lipoprotein particles.Question: What is the function of Hepatic Lipase? Answer: Hepatic Lipase degrades TGs remaining in intermediate-density lipoprotein (IDL) and chylomicron remnants.Question: What is the role of Hormone-sensitive Lipase? Answer: ...
Lipid Transport: A Comprehensive Review in Question and Answer Format
Переглядів 52Рік тому
usmleqa.com/?p=27734 Question: What is lipid transport in biochemistry? Answer: Lipid transport refers to the movement of lipids within cells and between cells in an organism. It is an important process that helps maintain the structural integrity of cell membranes and distribute lipids to where they are needed in the body.Question: What are lipids? Answer: Lipids are a diverse group of biomole...
Starvation: A Comprehensive Q&A Review for USMLE Step 1 Preparation
Переглядів 55Рік тому
usmleqa.com/?p=27732 Question: What happens to blood glucose levels during starvation in the first 3 days? Answer: During starvation in the first 3 days, blood glucose levels are maintained by hepatic glycogenolysis, adipose release of free fatty acids (FFA), and muscle and liver shifting fuel use from glucose to FFA. Hepatic gluconeogenesis from peripheral tissue lactate and alanine, as well a...
A Comprehensive Review of Fed & Fasting for USMLE Step 1 in Q&A Format
Переглядів 49Рік тому
usmleqa.com/?p=27730 Question: What are the priorities during Fed state (after a meal)? Answer: The priorities during Fed state (after a meal) are to supply sufficient glucose to the brain and RBCs and to preserve protein.Question: What processes occur during Fed state (after a meal)? Answer: During Fed state (after a meal), Glycolysis and aerobic respiration occur. Insulin stimulates the stora...
Metabolic Fuel Use: A Comprehensive Q&A Review
Переглядів 35Рік тому
Metabolic Fuel Use: A Comprehensive Q&A Review
Understand the Science Behind Fasted vs Fed State: A Detailed Explanation
Переглядів 106Рік тому
Understand the Science Behind Fasted vs Fed State: A Detailed Explanation
A Comprehensive Review of Fatty Acid Metabolism: Q&A Format
Переглядів 30Рік тому
A Comprehensive Review of Fatty Acid Metabolism: Q&A Format
Fatty Acid Metabolism: A Comprehensive Review in Question and Answer Format
Переглядів 73Рік тому
Fatty Acid Metabolism: A Comprehensive Review in Question and Answer Format
In-Depth Review of Hunter Syndrome and Lysosomal Storage Diseases: Essential Questions and Answers
Переглядів 44Рік тому
In-Depth Review of Hunter Syndrome and Lysosomal Storage Diseases: Essential Questions and Answers
Comprehensive Guide to Hurler Syndrome: Q&A Format
Переглядів 41Рік тому
Comprehensive Guide to Hurler Syndrome: Q&A Format
Niemann-Pick Disease: A Detailed Overview in Question and Answer Format
Переглядів 66Рік тому
Niemann-Pick Disease: A Detailed Overview in Question and Answer Format
In-Depth Review of Gaucher Disease: A Comprehensive Q&A Guide
Переглядів 54Рік тому
In-Depth Review of Gaucher Disease: A Comprehensive Q&A Guide
In-Depth Review on Krabbe Disease: A Q&A Guide for Understanding this Lysosomal Storage Disorder
Переглядів 77Рік тому
In-Depth Review on Krabbe Disease: A Q&A Guide for Understanding this Lysosomal Storage Disorder
Metachromatic Leukodystrophy: A Comprehensive Review in Q&A Format
Переглядів 40Рік тому
Metachromatic Leukodystrophy: A Comprehensive Review in Q&A Format
Expert Insight into Lysosomal Storage Diseases, Sphingolipidoses, and Fabry Disease: A Q&A Review
Переглядів 45Рік тому
Expert Insight into Lysosomal Storage Diseases, Sphingolipidoses, and Fabry Disease: A Q&A Review
Tay-Sachs Disease and Lysosomal Storage Diseases: A Comprehensive Review
Переглядів 156Рік тому
Tay-Sachs Disease and Lysosomal Storage Diseases: A Comprehensive Review
Lysosomal Storage Diseases: A Comprehensive Question and Answer Review
Переглядів 78Рік тому
Lysosomal Storage Diseases: A Comprehensive Question and Answer Review
Comprehensive Review of McArdle Disease (Type V): A Q&A Format Study Guide
Переглядів 441Рік тому
Comprehensive Review of McArdle Disease (Type V): A Q&A Format Study Guide
In-Depth Review of Andersen Disease (Type IV) - A Q&A Format for Medical Students
Переглядів 564Рік тому
In-Depth Review of Andersen Disease (Type IV) - A Q&A Format for Medical Students
دوست من سیرنگومیلی داره آیا همه بیماران در ناحیه دست و پا معلولیت پیدا میکنند؟ آیا کسی بیمار بوده و خوب شده؟ لطفا لطفا لطفا جواب بدید
ایا کسی هست درمان شده باشه؟😢
Thanks💯❤
I have a presentation on this disease next week
Red urine from myoglobinuria is not a symptom of the disease, it is a rare effect of over exertion. Most patients do not experience it often.
Thank you yes we need more research but this helps
LOL, until recently I hadn't realized that Mario updated his favorite way to tackle ED and it's a relief! Although what he previously suggested was pretty good, it was a real pain to follow... I just go'ogled the latest by Mario Volpstein, it's so much simpler and potent now!
Thanks man, really helps me review when i cant read or want to look at screen
Why did Toronto neurology ignore the enlarged ventricles shaped like devils horns on my MRI in about 2011? Did they just want me to keep having seizures? Is that why the word epileptic is so widely used and other symptoms like neck issues and pressure in head and eye ignored? Is it all in the name of medicine?
I appreciate your determination in helping me get rib of my Peyronie's disease naturally.Thank you Dr Emuakhe for your support, perseverance and love..
Being recommended to Dr Emuakhe on UA-cam was a blessing after years of suffering from Peyronie's disease,I have finally been cured,thank you doc..
thank you.
Thank you very much I got the real formula❤
kutte srf reading krte ho
How do I get my stomach flat again? I was thinking paracentesis.
McArdle does not mean “muscle”. The disease was named after Dr. Brian McArdle. The second wind phenomenon has more to do with fatty acid metabolism than increased blood flow.
This is truly the worst way to study for Step 1.
Metabolismmmm
Thanks
Thanks a lot. Its was helpful
Thanks
Thank you I loved how you wrote all the script in the description 🙏🏻❤️
All muscles in my body tighten up and I can't move for a short time. I'll try to stand but they freeze and getting up is like pushing a brick wall. Once up the muscles release and im able to walk. Sweat all the time, blurry vision, even sneezing causes entire bodies muscles to freeze
What’s pressure on the fingers and the value of the ability to read people? 6 months… oop let’s rewind ten years, honestly some of these doctors are really arrogantly rude; if they were from a 3rd world country I’d imagine them polishing their shiny brass buttons and showing their learned credentials, whilst having no moral fibre at all, actually quite shocking that old prison warden status of office hierarchy experiment. Yesterday I watched an excerpt of Elhers danios syndrome and wondered when private healthcare will be the only option for any ailments in the uk; no qualms for a crushing.
ua-cam.com/video/qwV8tp7381k/v-deo.htmlsi=tToBxpHHj0pzaMhp
Hey, I’ve purchased your videos on the website but I’m unable to access it, can anyone help me with that
Hi I subscribed for biochemistry but I don’t have access to the videos
ALERT !!!!!!! Don't click on the link, sounds fishy!!!! Why do I need to add an extension to my chrome browser just to check it out ???
It would be nice to add the medications names while you mentioned them to get a better visual
I thought cowdry a did not include cmv..cmv would be type b. Also in the intro it should state cowdry bodies can be eosinophilic or basophilic, not just eosinophilic.
Pls why ppp is low activity in muscle ,, pls give me answer
Pls explain inhibition of pyruvate carboxylase
❤❤❤❤❤ I love this
This might be the least helpful video I've ever watched
fr
My baby girl affected with i cell disease... now she is 1 1/2 year's old any treatment available for her??? Please tell me
👍👍👍👍👍
Whats systemic dilation
Unfortunately this is out of date
W
Please give
1:20 At last we got to know the pathogenesis of acute pancreatitis in case of hypertriglyceridemia !
Hello, I have a question, i need to know the role of s cdk on activation cdc25 i hope to get an answer as soon as possible thank u
Can a patient suffering from metamorphopsia be cured 🥺?
Nice video
No important concept explained, just a read to step 1
Great!
It is easy to understanding sir pls tell calculations also
❤
you just read from FA didnt explain !
Sir, Please make such videos for microbiology.